α-cd69 efluor 450 clone h1.2f3 antibody (Thermo Fisher)
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α-cd69 efluor 450 clone h1.2f3 antibody
α Cd69 Efluor 450 Clone H1.2f3 Antibody, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cd69+clone+h1%2E2f3+antibody/anti+cd69/pmc12002665-13-0-6
Average 90 stars, based on 1 article reviews
α Cd69 Efluor 450 Clone H1.2f3 Antibody, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cd69+clone+h1%2E2f3+antibody/anti+cd69/pmc12002665-13-0-6
Average 90 stars, based on 1 article reviews
α-cd69 efluor 450 clone h1.2f3 antibody - by Bioz Stars,
2026-10
90/100 stars
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1) Product Images from "Somatic Miwi2 modulates mitochondrial function in airway multiciliated cells and exacerbates influenza pathogenesis"
Article Title: Somatic Miwi2 modulates mitochondrial function in airway multiciliated cells and exacerbates influenza pathogenesis
Journal: iScience
doi: 10.1016/j.isci.2025.112291
Figure Legend Snippet:
Techniques Used: Blocking Assay, Virus, Recombinant
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Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Recombinant:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. 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Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), RNA Sequencing:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Software:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Chromatin Immunoprecipitation:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Flow Cytometry:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Magnetic Resonance Imaging:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Clone Assay:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Immunopeptidomics:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Staining:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Infection:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Activation Assay:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Expressing:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Quantitation Assay:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Labeling:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Two Tailed Test:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Injection:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Virus:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Recombinase Polymerase Amplification:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), Isolation:Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Ex vivo activation of the GCN2 pathway metabolically reprograms T cells, leading to enhanced adoptive cell therapy Article Snippet: Article Title: GOT1 constrains T H 17 cell differentiation, while promoting iT reg cell differentiation. Article Snippet: Because of a common requisite for TGFβ as a differentiating cytokine and the antagonizing effect of RORγt and FOXP3—master transcription factors for T helper 17 (TH17) and regulatory (Treg) cells, respectively—a reciprocal nature of TH17 and Treg cell differentiation often exists.. T. Xu et al. reported that pharmacological inhibition of glutamate oxaloacetate transaminase 1 (GOT1) by (aminooxy)acetic acid (AOA) metabolically reprogrammed TH17 cells towards induced Treg (iTreg) cells by altering the epigenetic landscape.. By contrast, here we show that the genetic deletion of Got1 in naive mouse CD4 T cells leads to enhanced IL-17A production associated with reduced FOXP3 expression under TH17-cell-polarizing conditions and mitigates FOXP3 expression in iTreg cells. Article Title: BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction Article Snippet: Cellular activation Peripheral blood and peritoneal leukocytes were isolated as described above, and cellular expression of Article Title: Lymph Node Conduits Transport Virions for Rapid T Cell Activation Article Snippet: Cells were stained with the following antibodies: Article Title: Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function Article Snippet: Antibodies used were: Article Title: Radiotherapy and CTLA-4 Blockade Shape the TCR Repertoire of Tumor-Infiltrating T Cells Article Snippet: A small fraction of sorted T cells was analyzed for Article Title: Prostate Cancer Cells Express More Androgen Receptor (AR) Following Androgen Deprivation, Improving Recognition by AR-Specific T Cells Article Snippet: Mouse antibodies: CD3 (clone 17A2, BD Biosciences), CD4 (clone GK1.5, BD Biosciences), CD8 (clone 53-6.7, BD Biosciences), CD45 (clone 30-F11, BD Biosciences), |
